Renal Colic

Urologiia. 2007 Jul-Aug;(4):20-3, 25.

Effects of magnetolaser radiation on pathologic process in the kidneys in experimental renal colic.

[Article in Russian]

Avdoshin VP, Andriukhin MI, Lebedeva TI, Shirshov VN.

The experiments were made on 22 rabbits with induced single or recurrent renal colic. Ten animals after renal colic were exposed to magnetolaser radiation. The histological examination of renal tissue demonstrated colic-related changes both in renal body corpuscle and ductules. Parenchimal edema, parabiotic processes in epitheliocytes and even necrosis of some cells were found. In case of recurrent colic the changes were severe, much more epitheliocytes were damaged, inflammatory leucocyte infiltration appeared. Magnetolaser radiation reduces the damage, eliminates leucocyte infiltration. Lymphocytic infiltrates formed in renal interstitium after two sessions of magnetolaser therapy indicate stimulation of cell immunity. The same processes develop also in the contralateral kidney. Their activity depends on the number of colics. Therefore, magnetolaser radiation has a protective action both on the isolated organ and body as a whole.

Urologiia. 2006 May-Jun;(3):47-50.

Influence of low intensive laser irradiation on ultrastructural changes in kidney tissue in experimental colic.

[Article in Russian]

Avdoshin VP, Andriukhin MI, Lebedeva TI, Taskinen IuI.

This investigation aimed at estimation of the influence of low-intensive laser irradiation on ultrastructural changes in renal tissue according to electronic microscopy data in case of experimental renal colic. The experiment was made on twenty rabbits with a single or recurrent colic. Ten animals after a single colic were exposed to low intensive laser therapy. Electronic microscopy showed that single renal colic is associated with marked ultrastructural changes in the proximal tubules. In recurrent colic the edema and destruction were more expressed in all nephron parts. Low intensive laser irradiation suppresses pathological processes in nephrocytes both after a single renal colic and after a recurrent renal colic; has a systemic effect on the body; stimulates compensatory processes in the contralateral kidney.